Guidelines
Teaching 17 Sep 2009 Wythenshawe Hospital
Gut Brain Axis
- Peter Paine (Hope)
- Narcotic Bowel Syndrome (NBS)
- Opioid induced hyperalgesia (OIH) eg allodynia
- Treatment for chronic pain
- Nortriptyline eg 50->150 on (better than Amitriptyline)
- SSRI: Duloxetine, Venlafaxine
- Clonidine (Sympatholytic)
- Lorazepam
- Hyoscine
- Cathartics: polyethylene glycol
- CBT
- Alvimopan (SR naloxone)
- [Pregabalin > Gabapentin]
- early involvement of pain team
- psychiatrist guided opioid withdrawal
- Bloating: FODMAPs; consider probiotics
- colonoscopy: 1:1000 perforation
Appetite and satiety
- John McLaughlin (Hope)
- enteroendocrine cells -> vagus -> brain: hypothalamus
- hypothalamus: also effects of leptin, ghrelin, insulin, ...
- enteroendocrine cells <- {chemical | microbial | physical} stimuli
- stomach: leptin, ghrelin, orexin
- upper small intestine: CCK, amandramide
- ileum + colon: GLP-1/-2, PYY
- eg: CCK: icrs satiety and delays gastric emptying
- CCK: only stimulated by fatty acids > C12 (ie only those that need bile acids for uptake; and solid at body temperature, compared to C11 or below [eg dairy products, coconut])
- nutrient-sensing receptors: bitter/sweet, fatty acid receptors (GPR40, GPR41, ...)
- C12 (compared to C10) icrs satiety and experimentally dcrs tolerance to water load (b/o delayed gastric emptying): therefore: "fatty starter"!
- hyperphagia:
- usu hedonistic + obesogenic environment;
- rarely leptin deficiency, MC4R mutations, Prader-Willi syndrome, hypothal tu
- bulimia
- anorexia: ..., alcohol abuse, ..., disinterest/depression
- palliative Tx: corticosteroids, megestrol, in development: s.c. Ghrelin analogues (i.e. "orexogenic")
- Nutrition: macronutrients, micronutrients, weight
- bariatric surgery: 'ileal brake': nutrients usu don't get there, but if they do, then icrd secretion of GLP-1, PYY, which stop transit, stop eating
Diabetic Gastroenteropathy (DGE)
- Piyara Begum (Res Fellow, Hope)
- DGE
- oesophageal dysmotility
- diabetic gastroparesis
- small intestinal dysfx
- colonic abnlties
- Diabetic autonomic neuropathy (DAN)
- 20-40% (50-60% if >25y duration)
- Argyll-Robertson pupil
- vagotomy: gastroparesis
- Corneal Confocal Microscopy: CCM (small C-fibres): dcrd nerve fibre density, dcrd nerve branch density, dcrd nerve fibre length = reproducible
- COMPASS=Composite Autonomic Symptoms Scale
- CASS = score from autonomic fx tests
- mechanisms of DAN
- axonal damage
- microvascular damage
- autoimmune neuropathy; ...
- gastric interstitial pacemaker cells of Cajal (ICC)
- Syndromes
- Dysphagia: NB: treat candida (trush, odynophagia, ...)
- N&V: NB: treat H pylori if present
- often on endoscopy: food residue (but NB: 30-50% diabetics have delayed gastric emptying, not necessarily with symptoms!)
- delayed/rapid emptying (see Camilleri'07NEJM:356)
- no trial evidence for Tx: small, frequent, liquid meals; rarely: post-pyloric feed
- Metoclopramide, Domperidone, Erythromycin (but tachyphylaxis); +- pregabalin, 5HT3; no benefit from botox into sphincter
- in development: Motilin, Ghrelin receptor agonists
Obesity
- Dr John New (Hope)
- OSA:
- Epworth Sleepiness Scale
- prevalence: ~50% if BMI>40;
- mort x2.7
- Diet vs Exercise: 1kg fat = 7500kcal
- =13 hamburgers
- =50 pints
- =130km run (if 70kg), =90km run (if 100kg)
- Orlistat/Sibutramine:
- unlikely to succeeed if no wt loss in 4 wks
- helps dieting (cf nicotine replacement)
- Orlistat
- best dietetic result (in that trial): -5kg in 12mth
- combined with Orlistat: -10kg in 12mths
- XENDOS trial: dcr obesity->dcrd DM; NB: lose 2kg (-5.7kg vs -3.6kg) dcrs DM incidence by 1/3!!
- 6.2% wt loss->-0.5% HbA1c; 5-10% wt loss->-0.95% HbA1c; >10% wt loss->-1.53% HbA1c
- Sibutramine:
- icrs BP by 2mm, need to see GP q2wks initially
- amphetamine derived: ie pt feels well
- STORM trial
- 600kcal deficit diet->-10% wt in 6/12
- Salford: max 800kcal + multivit diet, daily wt, then 1200kcal and icr until wt maintained
- best medical success: ~20%
- bariatric surgery:
- £7-9k
- NICE:
- first line if BMI>50
- also after other interventions for BMI>40, or BMI>35 + complications
- usu 18y+ age
- wt loss and mortality
- bypass: -35% wt [but maybe 70% fat mass!], mort 1:200
- band: -20%, mort 1:2000
- may need cor. angio and PCI beforehand
- stomach volume: 800ml->50ml
- "physical [as opposed to cognitive] behavioural therapy"
- SE:
- steatorrhoea with fat
- dumping with CHO
- saggy skin
- SOS=Swedish Obesity Study NEJM'07: mort at 15yr: 12% vs 9%, dcrs DM, Htn,
NAFLD
- Dr Das (Stepping Hill)
- prevalence ~1/3 #?
- 80-90% benign outcome
- decompensated cirrhosis from NAFLD: poor survival:
- similar to those with decompensated ALD and continued to drink: ~5% at 2yr
- whereas ALD and stopped drinking: ~60% at 2yr
- AST>2xULN: SMR=1.78, ALT>2xULN: SMR=1.51
- NAFLD: icrd use of FFA as myocyte energy -> effects on heart, etc; therefore a/w CHD
- "fast food hepatotoxicity": effect of high energy density food
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